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Mechanistically, impaired iron export—e.g., zinc-mediated inhibition of amyloid precursor protein (APP) ferroxidase activity—provides a route to local iron accumulation (Duce et al., 2010), while familial AD presenilin mutations confer selective vulnerability to ferroptosis in human neuronal models (Greenough et al., 2022)
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8 Pathophysiology of Hepatic Encephalopathy Studies investigating the pathophysiology of HE have historically focused on the accumulation of various toxins in the bloodstream and brains of animal models and patients with chronic liver disease and/or portal hypertension
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